目的：针对小切口非超声乳化白内障摘出术后早期高眼压进行临床观察与分析, 检测其发生率, 探讨其发生机制,以指导临床工作。 方法：对2011-01/06在我院行小切口非超声乳化白内障摘出术的116例116眼进行回顾性分析。 结果：患者116眼中有35眼（30.2%）术后早期出现眼压升高。眼压升高峰值期在术后6～24h, 最高56mmHg(1mmHg=0.133kPa)，高眼压的88.6%也发生在术后＞6～24h内，此时间段眼压升高发生率明显高于其他时段(P＜0.05) 。高眼压的62.9%为轻度升高（＜30mmHg）。 结论：小切口非超声乳化白内障摘出术后早期高眼压发生率较高，但大多为轻度增高。残留的黏弹剂或晶状体残余皮质堆积在房角上阻塞小梁网、术中或术后的眼内出血致变性的红细胞-血影细胞及吞噬了血红蛋白的巨噬细胞或其他物质如色素颗粒阻塞了小梁网，房水流出受阻为眼压升高最常见的原因。

AIM:To detect the incidence and mechanism of high intraocular pressure(IOP) at the early stage after small incision non-phacoemulsification cataract surgery. METHODS:Totally 116 patients (116 eyes) who had undergone small incision non-phacoemulsification cataract surgery in our hospital in the first half of 2011 were retrospectively analysed. RESULTS:Among the 116 eyes，postoperative IOP elevation occurred in 35 eyes (about 30.2%).The IOP reached the peak mostly 6 to 24 hours after the surgery, up to 56mmHg (1mmHg = 0.133kPa). 88.6% of high IOP occurred at the same time. And in this period the incidence of IOP elevation were significantly higher than in other periods (P<0.01). 62.9% of high IOP rose slightly, less than 30mmHg. CONCLUSION:The incidence of higy IOP at the early stage after small incision non-phacoemulsification cataract surgery is high, but most of the IOP rose slightly. The most common reason is that some substances blocked the trabecular meshwork. These substances were residual viscoelastic agent, residual cortex, denatured red blood cells, macrophages and pigment particle.These substances blocked the trabecular meshwork, and aqueous humor outflow was obstructed, and then the IOP rose. Other reasons also included IOL subluxation, endophthalmitis, etc.