多种眼部损伤可诱导角膜新生血管形成，促进疾病发展，造成角膜水肿、视力受损，甚至失明，因此抑制角膜新生血管有助于延缓疾病进程并降低角膜损伤，具有十分重要的临床意义。本文将对参与角膜新生血管形成的细胞及分子作最新的系统论述，并分析可能的抑制靶点，以期为科研及临床提供参考。

Various ocular injuries can induce corneal neovascularization, which promote the development of diseases, causing corneal edema, impaired vision and even blindness. Therefore, with very important clinical significance, inhibiting corneal neovascularization can help to delay the progression of diseases and reduce corneal damage. This article will make the latest systematic discussion on the cells and molecules involved in corneal neovascularization, and analyze the possible inhibitory targets, hoping to provide references for scientific research and clinical practice.

国家自然科学基金资助项目(No.81770899)